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Moreover, ER stress induced by NS5A and core HCV proteins causes calcium release from the ER into the cytoplasm

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Transgenic overexpression of bone morphogenetic protein 11 propeptide in skeleton enhances bone formation

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GCH1, independent of the GPX4 antioxidant pathway, primarily enhances the antioxidant activity of cells by promoting BH4 production and thus protects cells from ferroptosis
